Molecular programs induced by heat acclimation confer neuroprotection against TBI and hypoxic insults via cross-tolerance mechanisms

Neuroprotection following prolonged exposure to high ambient temperatures (heat acclimation HA) develops via altered molecular programs such as cross-tolerance (Heat Right Pedal Arm Acclimation -Neuroprotection Cross-Tolerance -HANCT).The mechanisms underlying cross-tolerance depend on enhanced on-demand protective pathways evolving during acclimat

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